By Shanda H. Blackmon, Min P. Kim, Karen J. Dickinson
This atlas offers a complete, state of the art evaluate of all interventions that pertain to the esophagus. It incorporates a assessment of the present staging modalities, ablation applied sciences, resection and reconstruction innovations, and illness class. Evidence-based directions concerning how each one intervention is selected also are integrated. With colour illustrations and images for every surgical procedure, the atlas info particular anatomic subject matters reminiscent of micro-anatomy of Barrett’s and Dysplasia, EMR pathology, endoscopic ultrasound, and standard surgical anatomy. every one intervention is gifted in activity structure as a role record to be checked-off as every one step is completed.
Written via specialists within the box, Atlas of Esophageal ailment and Intervention: A Multidisciplinary Approach serves as a necessary source for any practitioner who plays esophageal intervention and should advisor new surgeons and gastroenterologists into the hybrid multidisciplinary method of this disease.
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Extra info for Atlas of Esophageal Disease and Intervention: A Multidisciplinary Approach
Cytologic sampling of esophageal and gastroesophageal lesions is complementary to tissue biopsy. This chapter covers the basic gross features as well as the cyto- and histopathology of commonly encountered esophageal lesions. B. H. J. 1007/978-1-4939-3088-3_2 19 20 B. Gorman Normal Esophagus The innermost layer of the normal esophagus is composed of epithelium bounded by a basement membrane (Figs. 3). The epithelium rests on top of the lamina propria, which contains lymphatic channels. The muscularis mucosa is the deepest layer of the mucosa, beneath which is the submucosa.
Note the irregular size and shape of gland lumina with intraluminal necrosis. The tumor cell nuclei are large, with prominent nucleoli 36 Fig. 13 Mucinous adenocarcinoma (a recurrence). (a) H & E, ×40. Squamous epithelium overlies tumor cells in a background of mucin. (b) H & E, ×100. Irregular nests of tumor cells infiltrate the stroma. (c) H & E, ×200. (d) H & E, ×400. Note the signet ring appearance of several tumor cells. This is due to intracytoplasmic mucin. Although some of the tumor cell nuclei are small, they are hyperchromatic, angular, and displaced by the mucin vacuoles B.
6 (continued) 28 B. Gorman Fig. 7 (a) Barrett’s esophagus, indefinite for dysplasia (H & E, ×100). (b) Barrett’s esophagus, indefinite for dysplasia (H & E, ×400). The cells show cytologic features of dysplasia that include nuclear enlargement, hyperchromasia, and some nuclear stratification. Diagnostic features of high-grade dysplasia are not seen a b Nuclear enlargement, hyperchromasia and focal stratification Goblet cell Mitotic figure approaching gland lumen 2 Histologic Anatomy 29 Barrett’s Esophagus, Low-Grade Dysplasia Low-grade dysplasia is characterized by nuclear crowding, stratification, atypia, and increased mitotic figures (Fig.
Atlas of Esophageal Disease and Intervention: A Multidisciplinary Approach by Shanda H. Blackmon, Min P. Kim, Karen J. Dickinson